Dr. Robert Brickner is board certified in Internal Medicine, Endocrinology, Diabetes and Metabolism. He is also licensed, with over 30 years’ experience in the medical field. Dr. Brickner is an Assistant Professor of Internal Medicine with the UCF College of Medicine. Prior to this, he was the Chief of Endocrinology & Metabolism from 2005-2013 and a Physician Leader for the ADA Recognition of Diabetes Program from 2008-2011 with the Bay Pines VA Health Care System.
Dr. Brickner is a member of The American Association of Clinical Endocrinologists and The Endocrine Society. He served as Chairperson of the Research and Development Committee from 2010-2011 with the Bay Pines VA Health Care System. In 2000, Dr. Brickner was named one of Milwaukee Magazine’s “Top Doctors” in Endocrinology.
In partnership with the VA, UCF College of Medicine has identified individuals that provide a significant contribution to the college and medical students. Dr. Brickner joined the college in April of 2016 as part of this partnership. VA faculty members are invited to participate in teaching assignments, curriculum and admissions committees, student affairs and research mentorship. Their contributions make a significant impact for both the college and students.
- Brickner R, Raff H. The Renin-Angiotensin-Aldosterone System During Hypoxia: Is the Adrenal an Oxygen Sensor?” Hypoxia and Mountain Medicine. Edited by Sutton JR, Coates G, Houston CS. Queen City: Burlington, Vermont. p. 42-49. 1992.
- Shaker JL, Magill SB, Lalande BM, Brickner RC, Findling JW. Endocrine Manifestations of Celiac Disease. The Endocrinologist. Vol. 12, p. 110-116. 2002.
- Magill SB, Raff H, Shaker JL, Brickner RC, Knechtges TE, Kehoe ME, Findling JW. Comparison of Adrena Vein Sampling and Computed Tomography in the Differentiation of Primary Aldosteronism. Journal of Clinical Endocrinology and Metabolism. Vol. 86, No. 3, p. 1066-1071. 2001.
- Shaker JL, Brickner RC, Findling JW, Kelly TM, Rapp R, Rizk G, Haddad JG, Schatch DS, Shenker Y. Hypocalcemia and skeletal disease as presenting features of celiac disease. Archives of Internal Medicine. 157: p. 1013-1016. 1997.
- Findling JW, Shaker, JL Brickner RC, Riordan P, Aron DC. Curbside consultation in endocrinology: a prospective observational study. The Endocrinologist. 6:328-331. 1996.
- Shaker JL, Brickner RC, Divgi AB, Raff H, Findling JW. Renal phosphate wasting, syndrome of inappropriate antidiuretic hormone, and ectopic corticotrophin production in small cell carcinoma. American Journal of the Medical Sciences. p. 310:38-41. 1995.
- Brickner R, Raff H. The conversion of corticosterone to aldosterone is the site of the oxygen sensitivity of the adrenal zone glomerulosa. Endocrinology. 130, I, p 88-92. 1992.
- Brickner R, Raff H. Potassium-stimulated stimulated aldosteronogenesis is oxygen sensitive. Journal of Endocrinology. 129, p. 43-38. 1991.
- Findling JW, Kehoe ME, Shaker JL, Brickner RC, Raff H. Correlation of the interpetrosal sinus gradient with the location of ACTH-secreting micro adenomas. Program of the 4th International Pituitary Congress, P F9.
- Findling JW, Shaker JL, Brickner RC, Riordan P, Aron DC. Curbside consultation in endocrinology: a prospective observational study. The Endocrine Society, Program and Abstracts, No. P 1-72. June 1995.
- Shaker JL, Brickner RC, Kelly TM, Rapp R, Rizk G, Haddad JG, Schalch DS, and Shenker Y. Hypocalcemia and skeletal disease as primary clinical manifestations of celiac disease. The Endocrine Society, Program and Abstracts, No. P 2-620. June 1995.
- Brickner RC, Raff H, Shaker JL, Findling JW. Increased ACTH by immunoradiometric assay is an early indicator of primary adrenal insufficiency. Program of the 76th Annual Meeting of the Endocrine Society, P. 517. 1994.
- Shaker JL, Brickner RC, Divgi AB, Findling JW. Renal phosphate wasting associated with metastatic small cell carcinoma. Journal of Bone and Mineral Research, Program and Abstracts, P. s332. September 1992. (Adult Bone and Mineral Working Group).
- Shaker JL, Brickner RC, Sirus SR, Cerletty JM. Octreotide acetate-induced reduction of a large thyrotropin (TSH)-secreting pituitary tumor with correction of hyperthyroidism and hypopituitarism. The Endocrine Society, Program and Abstracts, No. 1138. June 1991.